αβγRöntgen+Climate, nuclear power plants, and taxonomy in Europe
αβγRöntgenUV+Climate
The concept Climate has to include, and includes a “αβγRöntgenUV+Climate” subdomain.
This has to be considered by the @EU_Comission, and all participating in the
#Taxonomie #taxonomy discussion.
The “αβγRöntgen+Climate” subdomain refers primarily to the weak nuclear force,
and to the electromagnetic force.
Climate aspects described by Sykuro MANABE, Klaus HASSELMANN, Giorgio PARISI, and others are related to gravitation, thermodynamics, and chemical aspects etc.,
too.
Yours Stefan Geier, Haidholzen
#ClubofRome
#Atomkraft #nuclearenergy #Fukushima #Tschernobyl
αβγRöntgenUV+Climate, 8.1.2022 (αβγRöntgen+Climate):
AntwortenLöschenAziz SANCAR's enormous inportant results justify to add UV to the climate subdomain abstract noun "αβγRöntgen+Climate"; of course the + includes more ... .
Please, see:
Mechanisms of DNA Repair by Photolyase and Excision Nuclease
Nobel Lecture, December 8, 2015
by Aziz Sancar
https://www.nobelprize.org/uploads/2018/06/sancar-lecture.pdf
or Wikipedia today: Nucleotide excision repair
"Nucleotide excision repair (NER) is a particularly important excision mechanism that removes DNA damage induced by ultraviolet light (UV). UV DNA damage results in bulky DNA adducts - these adducts are mostly thymine dimers and 6,4-photoproducts. Recognition of the damage leads to removal of a short single-stranded DNA segment that contains the lesion. The undamaged single-stranded DNA remains and DNA polymerase uses it as a template to synthesize a short complementary sequence. Final ligation to complete NER and form a double stranded DNA is carried out by DNA ligase. NER can be divided into two subpathways: global genomic NER (GG-NER or GGR) and transcription coupled NER (TC-NER or TCR). The two subpathways differ in how they recognize DNA damage but they share the same process for lesion incision, repair, and ligation.
The importance of NER is evidenced by the severe human diseases that result from in-born genetic mutations of NER proteins. Xeroderma pigmentosum and Cockayne's syndrome are two examples of NER associated diseases.Nucleotide excision repair (NER) is a particularly important excision mechanism that removes DNA damage induced by ultraviolet light (UV). UV DNA damage results in bulky DNA adducts - these adducts are mostly thymine dimers and 6,4-photoproducts. Recognition of the damage leads to removal of a short single-stranded DNA segment that contains the lesion. The undamaged single-stranded DNA remains and DNA polymerase uses it as a template to synthesize a short complementary sequence. Final ligation to complete NER and form a double stranded DNA is carried out by DNA ligase. NER can be divided into two subpathways: global genomic NER (GG-NER or GGR) and transcription coupled NER (TC-NER or TCR). The two subpathways differ in how they recognize DNA damage but they share the same process for lesion incision, repair, and ligation.
The importance of NER is evidenced by the severe human diseases that result from in-born genetic mutations of NER proteins. Xeroderma pigmentosum and Cockayne's syndrome are two examples of NER associated diseases."
Yours Stefan Geier, Haidholzen